Fauci under deposition in COVID-19 social media censorship case


New York Post Published originally on Rumble on November 23, 2022

Fauci under deposition in COVID-19 social media censorship case

Dr. Robert Malone is Still Fighting


Armstrong Economics Blog/Censorship Re-Posted Nov 25, 2022 by Martin Armstrong

QUESTION:
Message: Dear Martin,


Thank you for all of the insight that you make available to us on a daily basis.  I’m hoping that you might be able to provide an update to your April 5, 2022 blog, “Dr. Robert Malone vs WEF”?  At the time, the good Doctor was in the process of outing the various WEF world leaders and government scoundrels so as to make the list public.  Hopefully this is still in the works and you might be able to provide a timeline and manner in which this important information will be disseminated?
Thank you in advance for your valuable insight and guidance!
KD

REPLY: Dr. Robert Malone is still fighting to expose those responsible for COVID. He has appeared on Maria Zeee’s program many times, often on the same day that I am scheduled to speak. We are fighting for the same cause, with Malone decoding the science as I expose the economic consequences.

Dr. Robert Malone is one of the original inventors of mRNA and DNA vaccines. He holds numerous patents for gene delivery, formulation, and vaccines. He admitted that he originally believed that health organizations such as the CDC and NIH were in place to help the people. He quickly realized that the health organizations are merely government pawns that have been used to suppress and manipulate the people into taking dangerous vaccines.

The MSM crowd attempts to discredit Dr. Malone at every turn. His Wikipedia page (untrustworthy site) has been hacked, his social media accounts were permanently banned, and many major news publications wrote scathing reviews on the doctor for spreading “misinformation” despite him being one of the top experts in his field. “If they can remove my voice, my experience, my expertise – they win,” Malone stated.

Dr. Malone on attempts to silence him:

“The more I have expressed data-based concerns about what is happening with the vaccines, the US Federal and WHO responses, the more I have been censored, defamed, and subjected to various forms of character assassination by big tech and legacy media. I am not alone in being targeted. Mainstream media has attacked and censored me and other prominent physicians/scientists who do not recite the governmental narrative.  This has been developed into a standard process and deployed worldwide as a technique for suppressing physician dissent – quite literally hunting physicians deemed guilty of thoughtcrimes (such as questioning vaccine safety and effectiveness) or of the “sin” of treating patients with lifesaving drugs in an outpatient setting.”

Dr. Malone continues to speak out against vaccinations, and people are beginning to listen to his message.

G20 To Impose COVID Vaccine to Restrictions on International Travel


Armstrong Economics Blog/Economics Re-Posted Nov 25, 2022 by Martin Armstrong

The world masters dictating to us, the scum of the earth, have adopted under the pretense of the COVID vaccine the means to shut down migration and travel internationally. This will naturally further restrict global economic growth, and everywhere we turn, these people claiming to be world leaders are leading us into a cliff on the other side of 2032.

We were hoping to hold a WEC in Dubai where everyone can fly into without vaccines — an international reunion face to face. These leaders of the world’s largest economies at the Bali G20 drafted and signed a declaration in which the 20 countries agreed to adopt vaccine passports with the purported goal of promoting global travel and tourism. In fact, any country adopting this will have the opposite impact. I have resigned myself that I will never see Australia, New Zealand, or Europe ever again. Whatever these people can do to destroy the world economy, they are doing

Those who understand what is really taking place and that we are on the edge of global defaults post-2024 are not likely to take these vaccines. Hence, they are really trying to prevent movements that will overthrow governments. Just like communism fell in 1989, our Western economies will collapse, and they cannot prevent it. It will not necessitate massive civil unrest. People are not buying their long-term debt anymore, and when they cannot keep funding this insanity, they will collapse of their own accord. This is not some child’s game of blowing bubbles. We live in the real world.

Died Suddenly


Armstrong Economics Blog/Vaccine Re-Posted Nov 24, 2022 by Martin Armstrong

The question is why are those in government acting so recklessly? They need money that bad?

mRNA Injections Dosage is Probabilistic. Conformity with Label Cannot Be Verified.


By Sasha Latypova Posted originally on the conservative tree house on Aug. 18, 2022, 7:00 p.m.

Writer at Independent | mRNA Fraud – Regulatory and Manufacturing Investigations

Journalist Article

Pfizer’s mRNA injections are supplied in multiple dose vials.  There are several versions available today, however the most widely used is the “purple cap” vial.  This version has been supplied from the beginning of the injections roll out globally.  Both, the EUA BNT162b2 product and the “fully FDA approved” (but strangely unavailable in the US) version of COMIRNATY come in the purple cap format.  The image below shows both products.  Did you spot the difference?

If you are wondering whether the vials simply contain different amounts of finished drug product and different quantities of each ingredient to account for the different number of doses, I have the answer – they do not.  Indeed, it would be a bad pharmaceutical practice to have identical looking vials containing different dosage of the same product.   

Review of Pfizer’s Chemistry Manufacturing and Controls module from EMA materials obtained via a data leak at the end of 2020 revealed the following information about the ingredients, dosage, and the total volume of the finished product in the vials:

Clearly, this means a 0.45 mL of drug product, when diluted with 1.8 mL of saline is supposed to deliver 5 doses of 30 mcg of mRNA each.  The same dosage information, was provided in Pfizer’s briefing document for the FDA VRBPAC meeting on December 10, 2020, requesting the initial Emergency Use Authorization.

The label dosage information for Pfizer COMIRNATY currently available on the FDA website.  Section 11 lists the ingredients in text form.  The quantities per dose listed are the same as were provided in the FDA and EMA documents at the end of 2020.  The total supplied volume of product in the vial is also the same – 0.45ml and calls for the same amount of the dilutant to arrive at the individual doses. 

  • “COMIRNATY […] supplied as a frozen suspension in multiple dose vials with purple caps […]; each vial must be diluted with 1.8 mL of sterile 0.9% Sodium Chloride Injection, USP […]. Each 0.3 mL dose of COMIRNATY supplied in multiple dose vials with purple caps […]contains 30 mcg of a nucleoside-modified messenger RNA (mRNA) encoding the viral spike (S) glycoprotein of SARS-CoV-2. Each 0.3 mL dose of the COMIRNATY […] also includes the following ingredients: lipids (43 mg ((4-hydroxybutyl)azanediyl)bis(hexane-6,1-diyl)bis(2-hexyldecanoate), 0.05 mg 2-(polyethylene glycol 2000)-N,N-ditetradecylacetamide, 0.09 mg 1,2-distearoyl-sn-glycero-3-phosphocholine, and 0.2 mg cholesterol), 0.01 mg potassium chloride, 0.01 mg monobasic potassium phosphate, 0.36 mg sodium chloride, 0.07 mg dibasic sodium phosphate 20 dihydrate, and 6 mg sucrose. The diluent (sterile 0.9% Sodium Chloride Injection, USP) contributes an additional 2.16 mg sodium chloride per dose.”

The math clearly doesn’t add up: 225 mcg of the active mRNA substance in a vial equals to 50 mcg in a 5-dose vial and 38 mcg in a 6-dose one. 

The explanation of this apparent mystery is that the mRNA injection ingredients are specified by weight while the dose administration is by liquid volume.  No direct translation is possible.  At the injection administration site, the untrained and unsupervised vaccinators who, no doubt, will be thrown under the proverbial bus first when this scam fully unravels, are involved in the last phase of mRNA product manufacturing – the making of the individual doses, outside of any possibility of quality control by the pharmaceutical company.  If you are wondering if this violates the FD&C Act – yes, it does.   This is like the illicit drug dealers cutting cocaine by hand in a basement lab, however, much less precise.   

The vials, after dilution, contain 7.5 doses by liquid volume, and they always did.  Pfizer relabeled vials from 5 to 6 doses without making any changes to the product but rewarding themselves with 20% more revenue while selling the same vials with the same effective number of doses.  Genius.  The dose therefore is probabilistic.  Assuming the ingredients, and especially mRNA distribute absolutely evenly in the vial after someone manually injects saline into it, turns it over 10 times, and then leaves it in the fridge for 6 hours, then each dose may be 30 mcg of mRNA.  However, that’s a silly assumption.  Of course, they do not.  A technician draws a dose, lets the vial sit for 6 hours, draws some more doses, forgets it’s 6 hours, or forgets to put the vial into the fridge, and then decides that they too should make an extra buck by drawing the 7th dose from what is supposed to be only 5 or 6 doses – there are numerous scenarios that can be imagined (and are documented in VAERS database and other sources). 

mRNA and LNPs are known to be highly unstable and to degrade rapidly.  They also will not distribute evenly in a vial as this is a manually made water-fat mixture with fat tending to float to the top, especially after several hours.  The doses are extremely uneven in composition of ingredients, some will contain 50%+ more mRNA and would this introduce 5-6 trillion extra mRNA molecules in the injection which will distribute all over the body in minutes, rapidly make toxic spikes, and may kill a person quickly.  Pfizer’s internal pharmacovigilance report obtained by FOIA had thousands of severe adverse events and deaths documented in 2 months after roll-out with median onset below 48 hrs.  Some doses will end up containing mostly water.  We are all familiar with the fact that lots of people have no adverse events after the injections, while many thousands have died, got severely injured and permanently disabled by these injections.  My own educated guess on this topic is that the doses from freshly opened and stirred vials would be the deadliest ones.  The doses that were drawn from the vials that set on a shelf for a while, especially if the syringe is taking the liquid from the bottom of the container first will tend to be mostly harmless.

This was never tested by any regulator as no acceptance criteria for vials/doses exist.  Batches of the product are released based on self-declared testing of the bulk products by the manufacturer.  Nobody can know the composition of the shots as they are administered to people with catastrophic consequences.  These products are fraudulent by design and product conformity with its label cannot be verified.    

CDC admits they SH*T the bed, Amber Heard style | HPH #136


By Habibi Bros. Published originally on Rumble on August 17, 2022 

Siraj and Jay discuss the CDC admitting to f*cking up the COVID-19 pandemic response, Liz Cheney gets ousted from Congress, developments in the FBI raid of Trump’s home at Mar-A-Lago, and the one-year anniversary after the catastrophic Afghanistan withdrawal. It’s everything that makes you want to drink on Habibi Power Hour.

Is There Another FBI Spy In The Trump Orbit? (Ep. 1828) – The Dan Bongino Show


The Dan Bongino Show Published originally on Rumble on August 11, 2022 

Is there another spy in Trumps friends and associates? Or could this be a Trump set up?

Monkeypox is Not the Next Pandemic


Armstrong Economics Blog/Disease Re-Posted Aug 12, 2022 by Martin Armstrong

The Biden Administration declared a public health emergency over monkeypox. This is not an airborne virus, and it is fairly difficult to catch as skin-to-skin contact is the primary method of transmission. The woke media does not want this fact released, but the Centers for Disease Control and Prevention (CDC) has found that 99% of all cases were found in men, and 94% have had male sexual encounters.

Additionally, nearly 20% of gay men who fell ill admitted to having 10 or more partners in the three weeks before symptoms began. About 40% of those who fell ill reported having two to four partners, while 14% reported having five to nine partners. Around 38% admitted to participating in group sex.

This is more of a sexually transmitted disease and should be presented to the public as such. CDC guidance:

“Public health efforts should prioritize gay, bisexual, and other men who have sex with men, who are currently disproportionately affected, for prevention and testing, address equity, and minimize stigma, while maintaining vigilance for transmission in other populations.”

There is no need to stigmatize people for their sexual preferences or repeat problematic misinformation that spread during the 80s during the AIDS epidemic. However, there is no need to scare the general public into thinking that monkeypox is easily transmissible. If they care about health (they don’t), then they should be honest about the virus and educate the demographic mainly at risk.

Vaccine Mandates Illegal in Costa Rica


Armstrong Economics Blog/Disease Re-Posted Aug 11, 2022 by Martin Armstrong

Newly elected President Rodrigo Chaves has outlawed vaccine mandates. “Today vaccines are no longer mandatory and any action against someone who does not want to be vaccinated is a violation of the law,” the president stated. Although the president is vaccinated, he stated that was a health decision he personally made. It is now against the law in Costa Rica to force someone to be vaccinated against their will.

The president lifted numerous COVID restrictions when he came into power this May. He began by eliminating the mask mandate for everyone who does not work in healthcare. The Association of Physicians and Surgeons of Costa Rica, the Association of Pharmacists of Costa Rica, and the National Medical Union of Costa Rica spoke out against the president’s order to lift masks. He set out to lift the vaccine mandate in May too, but finally signed it into law this August.

This is another win for medical autonomy and a nation refusing to prolong the COVID fear-mongering propaganda that decimated the global economy.

Multisystem inflammatory syndrome in children (MIS-C) does NOT justify (at all!) their vaccination against SARS-CoV-2


Geert Vanden Bossche, DVM, PhD General Manager at Voice for Science and Solidarity | The biggest challenge in vaccinology: Countering immune evasion originally published on TS News on Aug. 5, 2022

MIS-C is a disease that may occur in school-age children two to six weeks after infection with SARS-CoV-2 (SC-2) virus. MIS-C is a post-infectious inflammatory condition which typically occurs after asymptomatic/mild SC-2 infection. Some children may need hospitalization because of inflammatory reactions in different organs. While the syndrome can be serious, the absolute risk for MIS-C is very low (about 6.5 per 100 000 person-years) and known to be increased in boys aged 5-11 years with foreign-born parents, asthma, obesity, and life-limiting condition (https://www.thelancet.com/action/showPdf?pii=S2666-7762%2822%2900137-5). 
MIS-C mostly resolves within a few days after timely and adequate (immunosuppressive) treatment. As the pandemic evolves and more infectious Omicron (sub)variants are now dominating the scene, MIS-C is occurring less frequently and with diminished severity of disease (https://jamanetwork.com/journals/jama/fullarticle/2792718). This evolution cannot be entirely explained by C-19 vaccination of young children as vaccine coverage rates in this age group are still very low (15% and 3% as recently reported in studies from Israel and the US, respectively; https://jamanetwork.com/journals/jama/fullarticle/2792718https://academic.oup.com/cid/advance-article/doi/10.1093/cid/ciac471/6605071).
It is therefore tempting to speculate that enhanced viral infectiousness and transmission significantly contribute to dampening the incidence rate and severity of MIS-C in young children. This suspicion is supported by a previous study in which the authors speculated that some of the risk factors they identified for MIS-C could be associated with enhanced disease transmission (e.g., in children with foreign-born parents or explaining the shift in age from 12-15 years down to 5-11 years amongst children with MIS-C throughout the pandemic (https://www.thelancet.com/action/showPdf?pii=S2666-7762%2822%2900137-5). 

To better understand the origin of the disease, and why children in particular are susceptible to contracting MIS-C, it is important to understand how the child’s innate immune system is educated and trained to combat infections with glycosylated viruses causing acute self-limiting viral infection (ASLVI; e.g., SC-2) or acute self-limiting viral disease (ASLVD).

The child’s innate immune system first learns to discern relevant pathogen-derived molecular patterns and discriminate them from self-derived motifs. Once the child’s natural killer (NK) cells have been  ‘educated[1]’ (pre-primed) to adequately sense and distinguish pathogen-derived self-mimicking peptides (PSMPs) from  self-derived self-mimicking peptides, presentation thereof in high density patterns may trigger epigenetic changes that imprint these NK cells with memory (so-called ‘training’ of NK cells). 

In young children who have cleared their maternal antibodies (around the age of 6 months), abundantly produced innate (sometimes called ‘natural’) antibodies (Abs) play a critical role in initiating active use of their own immune system. In these children, innate Abs are tasked with recognizing and binding free-circulating self-derived glycan motifs (e.g., decorating foreign-derived  [including pathogen-derived] or self-derived proteins) to potentiate[2] the presentation of repetitive patterns of foreign- or self-derived self-mimicking peptides on the surface of autologous somatic cells or antigen (Ag-)presenting cells (APCs). Glycosylation of self-proteins is an important mechanism for inducing T cell-mediated peripheral tolerance[3] and, not surprisingly, mimicked by several pathogens (e.g., glycosylated viruses) as a strategy to subvert the host immune system. As they decorate themselves with self-mimicking patterns of self-glycans, glycosylated viruses (e.g., corona virus [CoV], influenza virus, respiratory syncytial virus [RSV], measles, mumps, rubella, varicella virus,…) can be recognized and captured by innate Abs and thereby contribute to educating the child’s innate immune effector cells (i.e., NK cells).

As the child grows up, the functional capacity of their innate Abs gradually declines so that their immune system can progressively replace the ‘self’-sensing innate Ab capacity by a pool of pre-primed NK cells that can recognize pathogen-derived self-mimicking (i.e., ‘altered self’) motifs on virus-infected or otherwise pathologically altered host cells such as to kill those cells.
For as long as a child possesses an abundant functional capacity of innate Abs, glycosylated pathogens and self-ligands will be complexed by innate Abs to educate NK cells on how to distinguish ‘self’ from ‘non-self’.  This is how the innate immune system of the young child is thought to ‘adapt’ to the early-life extra-maternal environment where it must learn to rapidly sense peptide motifs that differ from self-peptides. This would enable NK cells to target and kill autologous host cells that are decorated with such ‘altered self’ peptides (e.g., infected, or otherwise pathologically altered host cells). 

Once NK cells are educated, the NK cell training process dictates their functional re-programming (https://www.frontiersin.org/articles/10.3389/fimmu.2018.01869/full). Training is thought to result from epigenetic alterations that are triggered by changes in the SC-2 infectious landscape and generate ‘adaptive’ or ‘memory-like’ NK cells. Adequate training of its first line of immune defense enables the child to mount protective natural immunity against SC-2 (and other glycosylated viruses/ pathogenic agents sharing the same PSMPs[4]) upon future exposure. This can already explain why prophylactic childhood vaccinations using live attenuated virus are very efficient at inducing natural immunity against measles, mumps, rubella, varicella and generating herd immunity−it’s only when they become infected with an antigenically ‘shifted’ (i.e., very different) variant that individuals who acquired natural immunity can still contract disease due to ADEI.  

However, depending on viral infectious pressure, it is perfectly possible, even for a young and healthy child, to become susceptible to productive infection upon exposure to glycosylated, ASLVI-enabling viruses that do not normally cause symptomatic infection in young children.

When young and healthy children become infected during an outbreak of a virus with a relatively low reproduction number (R0; e.g., infection with common cold coronavirus [CoV] or seasonal Flu; R0 < 2.5), they almost always develop asymptomatic or very mild infection. However, dominant circulation of more infectious CoV or Influenza virus variants can occasionally provoke cases of severe disease in children. It is reasonable to postulate that enhanced viral infectiousness raises the chance for a person to become re-infected shortly after a previous course of asymptomatic infection. This will increase the likelihood that immature, short-lived Ag-specific Abs[5] of relatively low affinity, which typically develop after asymptomatic/ mild infection, will still be present when that person becomes re-exposed to the virus. Because of their Ag-specificity, these Abs may outcompete the child’s innate polyspecific IgM Abs, which have an even lower affinity for the protein Ag that is responsible for initiation of infection (i.e., spike [S] protein in the case of CoV). Depending on their titer, these non-neutralizing, Ag-specific Abs can therefore prevent or at least diminish binding of innate Abs to the virus. Although these short-lived Abs cannot neutralize the virus, they can bind to it and enhance its infectiousness.

This is particularly problematic in young children as insufficient training of their NK cells prevents effective immune targeting of SC-2-infected cells expressing virus-derived self-mimicking peptides on their surface. NK cells that are largely ‘pathogen-inexperienced’ together with enhanced SC-2 infectiousness would entail enhanced susceptibility of young children to SC-2 infection (i.e., so called ‘Ab-dependent enhancement of viral infectiousness’; ADEI). Hence,  re-infection shortly after previous asymptomatic exposure will likely allow the virus to break through the cell-based innate immune system of young children and could potentially cause (severe) disease (https://pubmed.ncbi.nlm.nih.gov/33391280/).
However, as these post-infectious Abs wane rapidly (they are no longer detectable at about 8 weeks), only a limited number of children will become re-exposed to the circulating virus shortly after their previous productive exposure. This already explains why most cases of MIS-C occur between 2 and 6 weeks (on average 4 weeks) after the previous asymptomatic/ mild infection, presumably depending on the titer of the infection-enhancing Abs at the timepoint of re-exposure. It is therefore not surprising to also observe high variability in the severity of MIS-C disease. 

More infectious SC-2 variants may enable stronger stimulation of NK cells and thereby readily prime NK cell effector responses in young children; alternatively, more infectious SC-2 variants could increase the likelihood for viral re-exposure to occur in the presence of a relatively higher titer of infection-enhancing anti-S Abs. Both phenomena are likely to reduce the risk of MIS-C in young children

After many years of NK cell vaccine research (which I was unable to publish for intellectual property  reasons), I determined that the recruitment on MHC class I molecules of PSMPs into ‘non-self’ high-density arrays (situated outside of the MHC class I peptide-binding groove!) is what allows for activation and epigenetic imprinting (i.e., training) of cytotoxic NK cells that are capable of killing host cells that present such PSMPs on their surface (for example as a result of viral infection).
I postulate that strong stimulation by enhanced viral infectiousness could even obviate the need for cumulative triggering of NK cells (so-called ‘training’) in order for NK effector cells to become imprinted with memory. NK cells that have acquired a memory-like phenotype could readily eliminate host cells that are infected with relevant glycosylated pathogens. Enhanced viral infectiousness in the young child could allow productive SC-2 infection even in the presence of innate Abs and thereby enable ‘power training’ of pre-primed NK cells. Even though symptoms could still be mild, productive infection would have the capacity to substitute a single ‘power training’ event for regular, incremental training of functional NK cell responsiveness to pathogen-derived ligands. 
As full-fledged NK cell ‘priming’ towards PSMPs would therefore improve with enhanced viral infectiousness, C-19 unvaccinated children (and even some adolescents) who recently contracted mild disease would be equipped with innate immune memory while no longer developing infection-enhancing anti-S Abs (as shown in fig. 1). Alternatively, enhanced viral infectiousness leads to higher viral infection rates and thereby shortens the average time window for a person to become re-exposed after a previous asymptomatic SC-2 infection. A shortened window for re-exposure makes it more likely that the latter occurs in the presence of a relatively high titer of infection-enhancing Abs. It is not unreasonable to assume that the mechanism of naturally induced infection-enhancing Abs is similar to the one previous described for vaccine-induced anti-S Abs−a high enough concentration of these Abs would allow a subset of these Abs to bind to SC-2 virions tethered to dendritic cells and thereby exert a disease-mitigating effect (https://www.voiceforscienceandsolidarity.org/scientific-blog/predictions-gvb-on-evolution-c-19-pandemic + fig. 2).  

Based on the rationale explained above, one could easily understand how the prolonged C-19 pandemic and the enhanced frequency of repetitive waves of more infectious variants (e.g., Omicron) is likely to have a ‘power training’ effect on relevant NK cells of young C-19 unvaccinated children developing mild primary infection (i.e., overlapping with abundant functional capacity of innate Abs) or to provide a strong disease-mitigating adaptive immune response in those who recently contracted asymptomatic SC-2 infection. The latter would be protected from severe and even moderate disease by virtue of infection-enhancing Abs and cytotoxic CD8+ T cells, respectively (as illustrated in fig. 2). A further increase in viral infectiousness would not make young, previously asymptomatically infected children more susceptible to disease but rather increase their likelihood to develop productive SC-2 infection and generate effector memory NK cells or further expand those (as shown by the arrows in green in fig. 1).
With this understanding, it is not surprising that the advent of Omicron (sub)variants has led to a rapidly regressing incidence rate and severity of MIS-C (https://jamanetwork.com/journals/jama/fullarticle/2792718).

How does mass vaccination affect the child’s susceptibility to SC-2 infection?

As the mass vaccination program during this pandemic has led to the dominant circulation of more infectious SC-2 variants, it is not surprising to find that a few, young (C-19 unvaccinated) children contracted MIS-C and even needed hospitalization—this was an extremely rare event at the beginning of the pandemic. However, the mass vaccination program has provided immune escape variants characterized by a higher level of intrinsic viral infectiousness (e.g., of the delta variant) with a competitive advantage. The ensuing higher infection rate in the population (and in households!) therefore came with an additional likelihood for young children to become re-infected shortly after their previous asymptomatic infection. As previously described, the incidence rate of MIS-C is now waning as a result of enhanced innate immune training and mitigation of (severe) disease by more and more infectious SC-2 variants that have now become dominant. 

Vaccinating children against SC-2 is a colossal scientific blunder with potentially disastrous health consequences 

Parents should be adequately briefed about early signs and symptoms of MIS-C (https://www.hopkinsmedicine.org/health/conditions-and-diseases/coronavirus/misc-and-covid19-rare-inflammatory-syndrome-in-kids-and-teens) as the prognosis is very favorable upon timely and adequate treatment. However, the consequences of vaccinating young children with these replication-incompetent C-19 vaccines will be an unforgivable sin that will only lead to hospitalization and mortality rates that dwarf those highest observed for MIS-C (https://www.trialsitenews.com/a/intra-pandemic-vaccination-of-toddlers-with-non-replicating-antibody-based-vaccines-targeted-at-aslvi1-or-aslvd2-enabling-glycosylated-viruses-pr-66e8b959https://www.trialsitenews.com/a/epidemiologic-ramifications-and-global-health-consequences-of-the-c-19-mass-vaccination-experiment-a212bb47).

Notwithstanding the fact that these vaccines may–FOR NOW (!)–still protect against (severe) disease from SC-2 as well as from other ASLVI- or ASLVD-enabling glycosylated pathogens (https://www.trialsitenews.com/a/vaccination-of-vulnerable-groups-against-monkeypox-virus-mpv-in-a-highly-c-19-vaccinated-population-will-drive-adaptive-evolution-of-mpv-and-ignite-2db3eac6), enhanced adsorption or internalization of more infectious Omicron (sub) variants (e.g., BA.4, BA.5 and BA.2.12.1) onto or into tissue-resident dendritic cells dampens presentation of other, pathogen-derived antigens by these professional APCs while exhausting CD8+ T cells (as illustrated in fig. 2). This is highly likely to diminish the child’s immune defense against a multitude of microbial glycosylated pathogens and prevent peripheral tolerance, thereby putting them at higher risk  of contracting immunopathologies (https://www.trialsitenews.com/a/epidemiologic-ramifications-and-global-health-consequences-of-the-c-19-mass-vaccination-experiment-a212bb47).

More importantly, the currently circulating Omicron (sub)variants are already endowed with higher intrinsic virulence that–for now–is still kept in check by the virulence-inhibiting activity of infection-enhancing anti-S Abs (as reviewed in: https://www.voiceforscienceandsolidarity.org/scientific-blog/predictions-gvb-on-evolution-c-19-pandemic). We have already witnessed how more infectious variants developed resistance to potentially infection-neutralizing Abs induced by C-19 vaccines and there is little doubt that more virulent SC-2 lineages will manage a similar ‘trick’ to develop resistance to potentially virulence-‘neutralizing’ Abs (especially since repeated exposure to more infectious circulating Omicron (sub)variants will recall these vaccinal S-specific Abs and thereby ensure sustained immune pressure). When this happens, vaccinated infants and toddlers will be left with an adaptive immune system that does no longer protect them from severe C-19 disease and with NK cells that have not been trained due to prolonged suspension of their education (https://www.trialsitenews.com/a/intra-pandemic-vaccination-of-toddlers-with-non-replicating-antibody-based-vaccines-targeted-at-aslvi1-or-aslvd2-enabling-glycosylated-viruses-pr-66e8b959). Prolonged sidelining of the child’s innate Abs is thought to hamper the functional capability of cytotoxic NK effector cells to sense and target virus-derived, molecular self-mimicking peptides that are expressed on virus-infected host cells. As already reported, lack of innate immune education could dramatically impede the child’s capacity to generate natural immunity to SC-2 in particular as well as other ASLVI- or ASLVD-enabling glycosylated viruses in general (https://www.trialsitenews.com/a/epidemiologic-ramifications-and-global-health-consequences-of-the-c-19-mass-vaccination-experiment-a212bb47).

On the other hand, diminished complexation of foreign glycosylated ligands by innate Abs could render NK cells that lack self-MHC-I inhibitory receptors hyporesponsive to stimulatory receptor activation as a result of their chronic low-level stimulation by self-derived peptides. Because of the resulting diminished threshold of NK cell activation in the periphery, young C-19 vaccinated children would be prone to developing immunopathologies.
However, in the likely event that resistance to potentially virulence-‘neutralizing’ Abs develops, it can be expected that−given the high SC-2 infection rate− C-19 vaccinated infants and toddlers will primarily succumb to Ab-dependent enhancement of severe C-19 disease rather than to severe disease from any other circulating ASLVI or ASLVD or from immunopathology.   

Conclusion

Increased SARS-CoV-2 exposure results from dominant expansion of more infectious SC-2 variants, a phenomenon undeniably caused by the C-19 mass vaccination program. As suggested by the results from earlier studies, increased exposure to SC-2 together with a number of predisposing factors renders very few young children susceptible to developing MIS-C following a recent asymptomatic infection (https://www.thelancet.com/action/showPdf?pii=S2666-7762%2822%2900137-5).
There is no doubt that vaccinating children against SC-2 is a colossal blunder and merely places the child at high risk of severe health damage. MIS-C has not only a low incidence (which is further declining) but can also be successfully treated using conventional drug therapy. This contrasts with the protective effect of C-19 vaccination against MIS-C, which is temporary and leaves the young child at high risk of contracting Ab-dependent enhancement of severe disease upon future exposure to new SC-2 variants (which will dominantly emerge as a result of the current population-level immune pressure on viral virulence).
It is critical to understand that the high viral infection rate in highly C-19 vaccinated populations due to mass vaccination, rather than a lack of C-19 vaccination, is responsible for this phenomenon. There is therefore no single scientific rationale for vaccinating children against SC-2−exactly the contrary is true: C-19 vaccination of young children is highly likely to not only provoke a soaring incidence of severe disease and mortality due to immunopathology and other microbial diseases but ultimately also due to SC-2. 
Public health authorities are creating the illusion for parents that C-19 vaccines will protect their children, instead of educating them how to recognize early signs and symptoms of MIS-C in order to seek highly effective treatment for their child in due time. In addition, they seem to ignore that preserving natural immunity in young children is critical as it is the key pillar of herd immunity to ASLVIs, including SC-2.

Figures

Fig. 1: Upon exposure to more infectious SC-2 variants, young children may develop MIS-C as a result of re-exposure shortly after previous asymptomatic infection. However, as their infectiousness increases, new SC-2 variants may break through the child’s innate Ab-mediated protection and thereby cause mild infection that imprints its NK cells with memory and therefore dramatically boosts the child’s first line of immune defense. Alternatively, more infectious variants enable re-exposure in the presence of higher titers of short-lived infection-enhancing anti-S Abs. In the latter case, young children are protected from severe disease presumably because a subset of anti-S Abs can bind to SC-2 virions tethered to dendritic cells (see fig. 2), thereby inhibiting severe/systemic disease whereas sustained activation of CTLs (as a result of the infection-enhancing capacity of these Abs upon their binding to free virions) further mitigates C-19 disease. Both scenarios may be responsible for the observed reduction in the incidence rate and severity of MIS-C as the pandemic continues to evolve (indicated by “–“ and arrows in blue). Since the pandemic has now evolved highly infectious SC-2 variants (i.e., the new Omicron [sub]variants), viral exposure of young children is more and more likely to readily cause mild infection resulting in NK cell ‘power training’ and further expansion of effector memory NK cells upon a further increase in viral infection rates (indicated by “+” and arrows in green). This may ultimately prevent young children from developing MIS-C all together.  

Fig. 2 (from https://www.trialsitenews.com/a/epidemiologic-ramifications-and-global-health-consequences-of-the-c-19-mass-vaccination-experiment-a212bb47):
Acute, self-limiting viral infections that don’t lead to systemic/severe disease (and possibly death) are terminated by M(ajor) H(istocompatibility) C(omplex)-unrestricted, cytotoxic CD8+ T cells that have no memory and the activation of which is triggered by a universal, pathogen-nonspecific Tc epitope comprised within the spike (S) protein. Unless an infected person progresses to developing severe disease, this is what allows a fairly rapid recovery from disease after primary productive infection (and certainly before fully functional virus-neutralizing Abs peak) [according to 2a-2b-2c-2d pathway]. However, rather than stimulating de novo generation of new neutralizing Abs towards variants that escaped the neutralizing activity of vaccine-induced Abs, exposure of vaccinees to these immune escape variants will rapidly boost their declining titers of non-neutralizing, infection-enhancing Abs (those are directed against an antigenic site that is conserved within the N-STD of all SC-2 variants and has therefore a license to commit ‘antigenic sin’ once it has primed the host’s immune system).

In vaccinees with poor experience in fighting productive infection (and hence, poor training of their innate immune defense according to pathway 1a-1b-1c) prior to C-19 vaccination, infection-enhancing Abs[6] that are responsible for preventing severe disease by binding to DC-tethered virus (according to 3a-3b-3c-3d pathway) can synergize with strongly activated cytotoxic CD8+ Tc-mediated killing (3c’) to even prevent C-19 disease all together and hence, render vaccinees asymptomatic despite their high susceptibility to re-infection (B + C ?  D). As prevention of disease is not due to prevention of productive infection but to accelerated abrogation of infection, these vaccinees will continue to shed and transmit SC-2 upon re-infection.  Whereas innate immune effector cells are MHC-unrestricted and polyspecific (i.e., NK cells) and, therefore, don’t drive immune escape, the infection-enhancing-Abs are Ag-specific (i.e., S-specific) and – if produced at high enough titers and with high enough affinity by a large part of the population – will promote natural selection of immune escape variants that can resist  the virulence-inhibiting capacity of these Abs. This is because vaccinees cannot prevent productive viral infection; consequently, the immune pressure they exert on viral virulence is suboptimal in that it cannot prevent the expansion in prevalence of immune escape SC-2 variants that have the capacity to overcome this immune pressure. Resistance of viral variants to the virulence-inhibiting activity of infection-enhancing Abs will inevitably cause Ab-dependent enhancement of severe disease (ADESD).  




1.  Education of NK cells that lack self-MHC-I inhibitory receptors but are endowed with germline-encoded NK cell activation receptors dictates their functional capability to recognize self- or pathogen-derived self-mimicking ligands and mediate innate effector functions; https://www.frontiersin.org/articles/10.3389/fimmu.2018.01869/full

2.  Besides their neutralizing activity, natural/innate Abs can, indeed, serve as immune potentiators (‘natural
adjuvants’) to upregulate the presentation of antigens on cell surface-expressed MHC class I molecules
https://pubmed.ncbi.nlm.nih.gov/14502281/)

3.  https://onlinelibrary.wiley.com/doi/full/10.1038/icb.2008.48;
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3966069/

4.  Enveloped glycosylated viruses are critical to educate the child’s innate immune system in ways that allow
recognition and elimination of somatic cells expressing PSMPs (as a result of viral infection or other pathologic
alteration) which may otherwise induce tolerance (e.g., cancer cells) or provoke autoreactive or immune
inflammatory responses (i.e., causing autoimmune or hyperinflammatory disease, respectively).

5.  i.e., ‘spike protein-specific’ in case of SC-2

6.  As previously explained, the non-neutralizing, infection-enhancing Abs are currently hampering trans infection at the level of distant organs such as the lower respiratory tract; this is what’s currently exerting population-level immune pressure on viral virulence: https://www.voiceforscienceandsolidarity.org/scientific-blog/predictions-gvb-on-evolution-c-19-pandemic).